OVER 2000 NEAR DEATH EXPERIENCES FROM ENCEPHALOPATHY IGNORED AND BEATEN AND
RAPED SINCE 1979 WORLD WIDE MEDICINE INCLUDING BEHAVIOR MEDICINE TO CONFIR
M : INTEND BEAT & RAPE & IGNORE H.E. VICTIM INCLUDES "BEDRIDDEN ARE NO HELP
TO COMMUNITY & FAMILY AS ROTTING FETUSES"
How severe can hypoglycemic encephalopathy be ?? It has to be differentiate
d from Mad Cow Disease when severe enough ??
http://pubs.rsna.org/doi/full/10.1148/rg.311105041
"Overall incidence rates under the age of 45 range from (about 1 in 10,000
persons have ischemic attack and/or stroke) 7 to 15 in 100000 people/year f
or all stroke (ischaemic and haemorrhagic)" :
ABOUT 0.1% OF ALL STROKES AND ISCHEMIC ATTACKS ARE PERSONS UNDER AGE 45 :ht
tp://www.hindawi.com/journals/srt/2011/209370/
KENNETH MARTIN DOLAN VERY VERY VERY IRREGARDLESS OF HABIT VERY VERY VERY VE
RY VERY UNLIKELY TO HAVE VASCULAR DISEASE PRIOR AGE 30:
http://www.hindawi.com/journals/srt/2011/209370/
Explains patterns of lesions in differential diagnoses especially stroke :
http://appliedradiology.com/articles/stroke-differential-diagnosis-and-mimi
cs-part-2
CASE FOR KENNETH MARTIN DOLAN BEING NEAR DEATH ALL CHILDHOOD WITH HYPOGLYCE
MIA ENCEPHALOPATHY CONSTANT FROM 1979 UNTIL 1996 :
Match patterns : MR imaging of hypoglycemic encephalopathy: lesion distribu
tion and prognosis prediction by diffusion-weighted imaging.
HOW HORRIBLE (HE) HAS BEEN FOR KENNETH MARTIN DOLAN AND CAN BE :
http://link.springer.com/article/10.1007/s00415-012-6480-z#page-1ANDhttp://
www.ajnr.org/content/31/3/559.long
PATHWAY AND TREE IN LOGIC DIFFERENTIALS WHITE MATTER LESIONS ARE ONLY CAUSE
D BY HYPOGLYCEMIC ENCEPHALOPHATHY AND WITHOUT 100% AFFECT ON BEHAVIOR AND M
OTOR SKILLS IN AREAS OF BRAIN NEVER ASSOCIATED WITH BEHAVIOR AND MOTOR SKIL
LS AND WHERE 100% ACCURATE IS KENNETH MARTIN DOLAN IS DIFFERENT THAN 100 MI
LLION OF 100 MILLION MEDICAL PSYCHOSIS AND NEUROSIS ALL LIFE WHERE "LOOPHOL
E" CLAIMED DIFFERENT THAN 100 MILLION OF 100 MILLION MEDICAL PSYCHOSIS AND
NEUROSIS IN BRAIN VOLUME
100% RULE OUT OF :
1) BLOOD VESSEL DAMAGE FROM NECK UP ISCHEMIC ATTACK : LESIONS WHILE GENERAL
IZED LESIONS THROUGHOUT THE BRAIN ARE ONLY IN SPECIFIC BRAIN LOBES AND WOUL
D HAVE MANIFESTED AS "SYNCHRONIZED" BLOOD VESSEL BREAKAGES ALL AT SAME TIME
IMPOSSIBLY
2) MIGRAINE HEADACHES DAMAGE FROM NECK UP LESIONS WHILE GENERALIZED LESIONS
THROUGHOUT THE BRAIN ARE ONLY IN SPECIFIC BRAIN LOBES AND WOULD HAVE MANIF
ESTED AS "SYNCHRONIZED" BRAIN LESIONS ALL AT SAME TIME IMPOSSIBLY
3) DEMYLIATING PROCESS LIKE PARKINSON'S AND ALZHEIMER'S DISEASE AS WELL AS
MULTIPLE SCLEROSIS DAMAGE FROM NECK UP: LESIONS WHILE GENERALIZED LESIONS T
HROUGHOUT THE BRAIN ARE ONLY IN SPECIFIC BRAIN LOBES AND WOULD HAVE MANIFES
TED AS "SYNCHRONIZED" INITIATION WHITE MATTER LESIONS ALL AT SAME TIME IMPO
SSIBLY
4) DISEASE AND INFECTION WOULD HAVE BEEN GENERALIZED AND NEVER IN SAME AREA
S AND IN DIFFERENT LOBES AT RANDOM OR IF IN SPECIFIC AREAS OF BRAIN NEVER B
EEN IN DIFFERENT SPECIFIC AREAS OF BRAIN
5) IDIOPATHIC WHITE MATTER LESIONS WOULD HAVE BEEN GENERALIZED THROUGHOUT B
RAIN NOT LEFT SPECIFIC PATTERNS
6) HYPOGLYCEMIC ENCEPHALOPATHY WOULD HAVE LEFT PATTERNS OF SPECIFIC YET WIT
HIN SPECIFIC AREAS OF THE BRAIN BECAUSE LESIONS WOULD HAVE MANIFESTED NEARL
Y ALL AT ONCE
MIGRAINE BASED WHITE MATTER LESIONS :
OTE ON 2000 CT AND 2010 MRI ANY & ALL BLEMISHES IN REGIONS OF BRAIN NOT AFF
ECT INCLUDING PYRAMIDAL ANY MOTOR SKILLS AND/OR BEHAVIOR OF KENNETH MARTIN
DOLAN TO PRODUCE RESULTS AS SEEN IN RAPE PHOTO 100% TECHNICALITY FREE
CT BRAIN SCAN SUMMARY FEBRUARY 05 2001
IMPRESSION:
No acute intracranial process.
Unenhanced head CT scan demonstrates no mass effect or shift.
MRI BRAIN SCAN SUMMARY JUNE 11 2010
(CONGRUENT WITH "OLD T2-FLAIR" OF "SCARS" OF HYPOGLYCEMIA ENCEPHALOPATHY)
NOTE : SUMMARY ONLY ALSO TRAUMA AS REFERRED WOULD HAVE BEEN THROUGH DIRECT
CONTACT OUTSIDE OF TECHNOLOGY USE ONLY LIKE BLUNT OBJECT OR FIST ETC.:
Addendium Begins:
Request was made by the patient to comment on the presence of evidence of t
rauma, including areas of hemorrhage and scarring in the brain.
There is no evidence of hemorrhage in this exam, specifically there is no e
vidence of diffuse axonal injury or other patterns of post traumatic hemorr
hage.
The few nonspecific foci of superatentorial white matter signal abnormality
are nonspecific however could conceivably represent small areas of gliosis
. However, these are a common finding seen in both symptomatic and asymptom
atic individuals. No patterns of encephalomalacia and gliosis that would be
seen in the setting of trauma.
Addendum Ends.
MRI brain without contrast.
Indication : Dizziness, lightheadedness, intracranial hemorrhage, headaches
,.
Techniques: Multiplanar, multisequence MR images of the brain were obtained
without contrast.
Findings : No hydrocephalus and basal cistems are patent. No mass effect or
midline shift. No intracranial hemorrhages or extra-axial fluid collection
s. Gradient echo imaging is negative. There are a few scattered foci of hyp
erintense T2/FLAIR abnormality in the subcortical and deep white matter, vi
sualized on images 15 and 16 of series 6. No other significant areas of abn
ormal signal in the brain parenchyma.
Cerebellar tonsils are well located. Midline structures are intact. Major v
ascular flow-voids are maintained. Orbits are unremarkable. Paranasal sinus
es and mastoid air cells are well aerated. Bones are within normal limits.
Impression:
1. A few nonspecific foci of supratentorial white matter signal abnormalcy.
This is a nonspecific finding that can be seen in both symptomatic and asy
mptomatic individuals. This can also represent chronic small vessel ischemi
a, a demyelinating process, infectious lesions (such as Lyme disease), sarc
oidosis, and has been reported in migraine disorders. Clinical correlation.
2. Otherwise unremarkable noncontrast MRI of the brain.
How severe can hypoglycemic encephalopathy be ?? It has to be differentiate
d from Mad Cow Disease when severe enough ??
http://pubs.rsna.org/doi/full/10.1148/rg.311105041
Unlike white matter lesions in brain of Kenneth Martin Dolan migraines are
found > 85% of the time in frontal lobe :
RESULTS:
One hundred and eighty-five patients (77% women) were included. Aura sympto
ms were classified as visual in 172 (99%) patients, sensory in 76 (42%), ap
hasic in 54 (30%), motor in 39 (21%) and vertebrobasilar in 17 (9%) patient
s. One hundred and four patients (57%) exhibited more than one type of aura
. D-WMLs were mainly detected in the frontal lobes (86%). There was no asso
ciation between type of aura and the presence of WMLs in any cerebral locat
ion.
https://www.ncbi.nlm.nih.gov/pubmed/20647177
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3241741/
Six of 46 (13%) of the migraineurs had white matter lesions versus three of
69 (4.3%) of the controls. The white matter lesions in migraineurs were s
een in a younger age group than in the controls. These findings agree with
recent MRI studies. Ischemia or an immune-based white matter demyelinatio
n are possible mechanisms for the white matter lesions.
http://chicagoheadacheclinic.com/archives/mri_migraineurs.html
BRAIN AND STRUCTURE VOLUME CHANGES IN MIGRAINE HEADACHE GIVING KENNETH MART
IN DOLAN 0% CHANCE OF MIGRAINE HEADACHES :
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3795609/
Migraine and Depression Linked to Smaller Brain Size
http://www.headaches.org/2013/06/15/migraine-and-depression-linked-to-small
er-brain-size/
Results
IHLs were identified in 1 of 140 controls (0.7%) and 13 of
295 migraineurs (4.4%; P0.04), slightly more in MA (8 of
161; 5.0%) than in MO (5 of 134; 3.7%; Table). All subjects
had a normal standard neurological examination and no
history of transient ischemic attack or stroke
http://stroke.ahajournals.org/content/strokeaha/37/4/1109.full.pdf
Inflammatory demyelinating diseases
https://en.wikipedia.org/wiki/Inflammatory_demyelinating_diseases_of_the_ce
ntral_nervous_system
WML distribution in Inflammatory demyelinating diseases
Ruled out : Idiopathic hypoglycemia
https://en.wikipedia.org/wiki/Idiopathic_hypoglycemia
Interesting note about radiology versus neuroscience :
Results
Inter-rater reliability was substantial-almost perfect between neuropatholo
gists (kappa 0.71 - 0.79) and fair-moderate between radiologists (kappa 0.3
4 - 0.42). Discriminating low versus high lesion scores, radiologic compare
d to neuropathologic evaluation had sensitivity / specificity of 0.83 / 0.4
7 for periventricular and 0.44 / 0.88 for deep white matter lesions. T2/FLA
IR WMHs overestimate neuropathologically confirmed demyelination in the per
iventricular (p < 0.001) areas but underestimates it in the deep WM (0 < 0.
05). In a subset of 14 cases with prominent perivascular WMH, no correspond
ing demyelination was found in 12 cases.
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3893472/
(MS) Multiple Sclerosis
HLA-DR2 and White Matter Lesion Distribution in MS
https://www.researchgate.net/publication/5369970_HLA-DR2_and_white_matter_l
esion_distribution_in_MS
Measuring Gray Matter and White Matter Damage in MS:
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4362212/
Distribution of Brain Sodium Accumulation Correlates with Disability in Mul
tiple Sclerosis: A Cross-sectional 23Na MR Imaging Study
http://pubs.rsna.org/doi/full/10.1148/radiol.12112680
http://journals.plos.org/plosone/article/file?id=10.1371/journal.pone.010
7263&type=printable
Increase of sodium channels in demyelinated lesions of multiple sclerosis
http://lnc.univ-amu.fr/IMG/pdf/1991_moll_na_chan_slerose.pdf
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2811971/
https://braindiseases.wordpress.com/2008/05/15/mri-white-matter-lesions-doe
s-it-represent-ms/
Neuro-Lyme Disease: MR Imaging
Findings1
http://pubs.rsna.org/doi/pdf/10.1148/radiol.2531081103
Have but need further rule out :
Sarcoidosis :
http://www.everydayhealth.com/sarcoidosis/guide/
Cutaneous Sarcoidosis
https://www.google.com/search?q=Cutaneous+Sarcoidosis&oq=Cutaneous+Sarc
oidosis&aqs=chrome..69i57.1002j0j1&sourceid=chrome&ie=UTF-8
A Retrospective Cohort Study of Lesion Distribution of HIV-1 Infection Pati
ents With Cryptococcal Meningoencephalitis on MRI
STROKE/ ISCHEMIC ATTACK RULE OUT :
MATHEMATICAL IMPOSSIBLE FOR KENNETH MARTIN DOLAN TO HAVE ISCHEMIC ATTACK AN
D/OR STROKE < 2000 :
KENNETH MARTIN DOLAN VERY VERY VERY IRREGARDLESS OF HABIT VERY VERY VERY VE
RY VERY UNLIKELY TO HAVE VASCULAR DISEASE PRIOR AGE 30: ABOUT 0.1% OF ALL S
TROKES AND ISCHEMIC ATTACKS ARE PERSONS UNDER AGE 45: "Overall incidence ra
tes under the age of 45 range from (about 1 in 10,000 persons have ischemic
attack and/or stroke) 7 to 15 in 100000 people/year for all stroke (ischae
mic and haemorrhagic)" :
http://www.hindawi.com/journals/srt/2011/209370/
Conclusion— Elderly people with silent brain infarcts and white mat
ter lesions are at a strongly increased risk of stroke, which could not be
explained by the major stroke risk factors.
Hypoglycemic encephalopathy: a case series and literature review on outcome
determination
http://link.springer.com/article/10.1007/s00415-012-6480-z#page-1ANDhttp://
www.ajnr.org/content/31/3/559.long
Very different than Kenneth Martin Dolan :
http://europepmc.org/articles/pmc4737263
Extent and Distribution of White Matter Hyperintensities in
Stroke Patients
The Sydney Stroke Study
Wei Wen, PhD; Perminder S. Sachdev, MD, PhD, FRANZCP :
https://pdfs.semanticscholar.org/04cb/13be8c40aabc29200ebe8303da707a1a8c5e.
pdf
Most white matter hyperintensities were in centrum semiovale (except for th
e area affected by the acute symptomatic infarcts), external capsules, basa
l ganglia, and brainstem, with little overlap with the acute symptomatic in
farcts (analysis of variance, P < 0·01).
MATCH THE BLOOD GLUCOSE MULTIPLE TESTS TO HYPOGLYCEMIC ENCEPHALOPATHY THAT
KENNETH MARTIN DOLAN TOLD "LIES AND CONSPIRES" (INCLUDING MEDICINE INCLUDIN
G NURSING MASS POPULATION PARANOID DISORDER AS KENNETH MARTIN DOLAN IS "TRA
NSFERENCE (OVERLAP OWN PARANOID PROBLEMS UPON KENNETH MARTIN DOLAN FROM EXT
ERNAL FROM KENNETH MARTIN DOLAN MARKERS : WANTING PSYCHIATRIC HOSPITALIZATI
ON OF 0% PSYCHOSIS AND 0% "GOING TO JAIL" AS "SICK" " AND COUNTERPARANOID (
REF : Pa 51 62 1987 2004 MMPI PFC >0.5% VOLUME MASS THAN PARANOIDS )
REPORT TIME 07/03/1987 14:40 3-8669210/0001 ROUTE TO : MOFFAT F L (DR. MOFF
AT 1986)
SPEC. TIME BLOOD, URINE COLL. TIME 07/03/187 1045 (2426709)
FASTING (MG/DL): 88
1/2 HOUR (MG/DL) : 101
1 HOUR (MG/DL) : 77
2 HOUR (MG/DL) : 76
3 HOUR(MG/DL) : 51
SPEC. TIME BLOOD, URINE COLL. TIME 07/03/187 1045 (2426709)
FASTING (MG/DL): 88 1/2 HOUR (MG/DL) : 101 1 HOUR (MG/DL) : 77 2 HOUR (MG/D
L) : 76 3 HOUR(MG/DL) : 51
http://link.springer.com/article/10.1007/s00415-012-6480-z#page-1ANDhttp://
www.ajnr.org/content/31/3/559.long
"Neuropathologic studies have demonstrated that the cerebral cortex, hippoc
ampus, and BG (Basil Ganglia) are most affected in severe hypoglycemia, how
ever, the brain stem and cerebellum are usually spared."
NOTE IN BRAIN OF KENNETH MARTIN DOLAN MOST LESIONS HAVE BEEN RESOLVED PRIOR
2001 AS SEEN ALSO IN CT AND MRI ABSENT OF BRAIN SAC INFLAMMATION : T2 FLAI
RS MUST BE FROM PRIOR 2001 AS UNRESOLVED LESIONS
http://www.ajnr.org/content/31/3/559.full
https://books.google.com/books?id=Pmcy24y2HyMC&pg=PA241&lpg=PA241&dq
=WHITE+MATTER+LESIONS+HYPOGLYCEMIA++DEEP&source=bl&ots=FQTz3Ut3D7&sig
=avDi7G0j8YeFz1SMSRMmmgFhh0E&hl=en&sa=X&ved=0CDsQ6AEwA2oVChMI-8XV0M
eExgIVxzasCh2heQAe#v=onepage&q=WHITE%20MATTER%20LESIONS%20HYPOGLYCEMIA%
20%20DEEP&f=false
RESOLUTION OF MOST LESIONS REVERSAL "IN ACTION"
http://www.jmnn.org/article.asp?issn=2278-1870;year=2014;volume=3;iss
ue=2;spage=102;epage=105;aulast=Thakur
https://inis.iaea.org/search/search.aspx?orig_q=RN:40102721
AGAIN A MATCH :
"In some cases of milder, reversible hypoglycemia, transient and isolated w
hite matter abnormalities involving the splenium of the corpus callosum, in
ternal capsules, and corona radiata have been reported, with patients makin
g a full recovery without neurologic deficit"
See ischemic vessel disease rule out elsewhere :
Grey matter not white matter is typically target of ischemic WML
"In adults, mild HIE may affect only the watershed zones. Severe HIE charac
teristically affects the gray matter structures, including the cerebral cor
tex, basal ganglia, and hippocampi. The thalamus and cerebellum may also be
affected, but the brainstem and cerebral white matter are typically spared
."
http://pubs.rsna.org/doi/full/10.1148/rg.311105041
The absence of localized hemorrhages on MR images in hypoglycemic encephalo
pathy is in marked contrast to the presence of regional minor hemorrhages i
n postischemic-anoxic encephalopathy.
http://stroke.ahajournals.org/content/28/3/584.full
http://www.ncbi.nlm.nih.gov/pubmed/23820485
http://onlinelibrary.wiley.com/doi/10.1111/j.1440-1789.2006.00656.x/abstrac
t;jsessionid=4EDBE3FF4A19EA682AD5D2725A9A40A0.f03t01?userIsAuthenticated
=false&deniedAccessCustomisedMessage=
http://neuropathology-web.org/chapter2/chapter2aHIE.html
http://www.ncbi.nlm.nih.gov/pubmed/1277057
http://www.ncbi.nlm.nih.gov/books/NBK28273/
White matter distribution in the elderly :
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2593803/
46 longitudinal studies evaluated the association of white matter hyperinte
nsities with risk of stroke (n=12), cognitive decline (n=19), dementia
(n=17), and death (n=10). 22 studies could be included in a meta-analys
is (nine of stroke, nine of dementia, eight of death). White matter hyperin
tensities were associated with an increased risk of stroke (hazard ratio 3.
3, 95% confidence interval 2.6 to 4.4), dementia (1.9, 1.3 to 2.8), and dea
th (2.0, 1.6 to 2.7). An association of white matter hyperintensities with
a faster decline in global cognitive performance, executive function, and p
rocessing speed was also suggested.
http://www.bmj.com/content/341/bmj.c3666
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